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Journal Cover Nature Communications
  [SJR: 6.539]   [H-I: 114]   [202 followers]  Follow
    
  This is an Open Access Journal Open Access journal
   ISSN (Online) 2041-1723
   Published by NPG Homepage  [136 journals]
  • Gli1 identifies osteogenic progenitors for bone formation and fracture
           repair
    • Gli1 identifies osteogenic progenitors for bone formation and fracture repair

      Gli1 identifies osteogenic progenitors for bone formation and fracture repair, Published online: 11 December 2017; doi:10.1038/s41467-017-02171-2

      Skeletal progenitors in postnatal mice are highly heterogeneous. Using lineage tracing and RNA-seq the authors show that Gli1+ cells give rise to all osteoblasts in mice, including those required for healing of bone fractures.Gli1 identifies osteogenic progenitors for bone formation and fracture repair, Published online: 2017-12-11; doi:10.1038/s41467-017-02171-22017-12-11
      DOI: 10.1038/s41467-017-02171-2
       
  • Functional reduction in pollination through herbivore-induced pollinator
           limitation and its potential in mutualist communities
    • Functional reduction in pollination through herbivore-induced pollinator limitation and its potential in mutualist communities

      Functional reduction in pollination through herbivore-induced pollinator limitation and its potential in mutualist communities, Published online: 11 December 2017; doi:10.1038/s41467-017-02072-4

      Herbivory-induced volatile organic compounds reduce pollinator visits to plants. Here, Glaum and Kessler show that despite reducing individual fitness, herbivore-induced changes can indirectly benefit overall community resilience, explaining species persistence in antagonized mutualistic communities.Functional reduction in pollination through herbivore-induced pollinator limitation and its potential in mutualist communities, Published online: 2017-12-11; doi:10.1038/s41467-017-02072-42017-12-11
      DOI: 10.1038/s41467-017-02072-4
       
  • Direction of actin flow dictates integrin LFA-1 orientation during
           leukocyte migration
    • Direction of actin flow dictates integrin LFA-1 orientation during leukocyte migration

      Direction of actin flow dictates integrin LFA-1 orientation during leukocyte migration, Published online: 11 December 2017; doi:10.1038/s41467-017-01848-y

      Integrin αβ heterodimer cell surface receptors mediate adhesive interactions that provide traction for cell migration. Here the authors show that actin flow can orient cell surface integrins during leukocyte migration, suggesting integrin activation by cytoskeletal force.Direction of actin flow dictates integrin LFA-1 orientation during leukocyte migration, Published online: 2017-12-11; doi:10.1038/s41467-017-01848-y2017-12-11
      DOI: 10.1038/s41467-017-01848-y
       
  • Identification of regulatory targets for the bacterial Nus factor complex
    • Identification of regulatory targets for the bacterial Nus factor complex

      Identification of regulatory targets for the bacterial Nus factor complex, Published online: 11 December 2017; doi:10.1038/s41467-017-02124-9

      The Nus factor complex regulates rRNA folding and prevents Rho-dependent transcription termination in bacteria. Here, Baniulyte et al. show that it also inhibits translation of one of the Nus factor-encoding genes, suhB, and probably regulates the expression of other genes in diverse bacterial species.Identification of regulatory targets for the bacterial Nus factor complex, Published online: 2017-12-11; doi:10.1038/s41467-017-02124-92017-12-11
      DOI: 10.1038/s41467-017-02124-9
       
  • Migratory dendritic cells acquire and present lymphatic endothelial
           cell-archived antigens during lymph node contraction
    • Migratory dendritic cells acquire and present lymphatic endothelial cell-archived antigens during lymph node contraction

      Migratory dendritic cells acquire and present lymphatic endothelial cell-archived antigens during lymph node contraction, Published online: 11 December 2017; doi:10.1038/s41467-017-02247-z

      Viral infection and vaccination both induce lasting persistence of antigens for protective responses. Here the authors show that migratory dendritic cells, independent of the transcription factor BatF3 for their development, contribute to “archived antigen” exchange with lymphatic endothelial cells.Migratory dendritic cells acquire and present lymphatic endothelial cell-archived antigens during lymph node contraction, Published online: 2017-12-11; doi:10.1038/s41467-017-02247-z2017-12-11
      DOI: 10.1038/s41467-017-02247-z
       
  • Pseudogap phase of cuprate superconductors confined by Fermi surface
           topology
    • Pseudogap phase of cuprate superconductors confined by Fermi surface topology

      Pseudogap phase of cuprate superconductors confined by Fermi surface topology, Published online: 11 December 2017; doi:10.1038/s41467-017-02122-x

      High-temperature superconductors exhibit pseudogap behaviour that remains of unknown origin, despite many years of intensive study. Here the authors study the onset of the pseudogap under pressure, providing evidence that it requires a hole-like Fermi surface and constraining future theoretical developments.Pseudogap phase of cuprate superconductors confined by Fermi surface topology, Published online: 2017-12-11; doi:10.1038/s41467-017-02122-x2017-12-11
      DOI: 10.1038/s41467-017-02122-x
       
  • Loss of PBRM1 rescues VHL dependent replication stress to
           promote renal carcinogenesis
    • Loss of PBRM1 rescues VHL dependent replication stress to promote renal carcinogenesis

      Loss of PBRM1 rescues VHL dependent replication stress to promote renal carcinogenesis, Published online: 11 December 2017; doi:10.1038/s41467-017-02245-1

      Mutations in VHL have been linked to clear cell renal cancer, but the molecular mechanisms involved remain unclear. Here the authors generate a mouse model closely mimicking the human disease and show that VHL loss induces DNA replication stress that is rescued by the concomitant loss of PBRM1 permitting transformation.Loss of PBRM1 rescues VHL dependent replication stress to promote renal carcinogenesis, Published online: 2017-12-11; doi:10.1038/s41467-017-02245-12017-12-11
      DOI: 10.1038/s41467-017-02245-1
       
  • Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and
           ubiquitination in DNA double-strand break repair
    • Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair

      Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair, Published online: 11 December 2017; doi:10.1038/s41467-017-02146-3

      DNA double-strand break (DSB) repair is a tightly regulated process that can occur via non-homologous end joining (NHEJ) or homologous recombination (HR). Here, the authors investigate how RECQL4 modulates DSB repair pathway choice by differentially regulating NHEJ and HR in a cell cycle-dependent manner.Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair, Published online: 2017-12-11; doi:10.1038/s41467-017-02146-32017-12-11
      DOI: 10.1038/s41467-017-02146-3
       
 
 
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