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Journal Cover Nature Medicine
  [SJR: 13.959]   [H-I: 439]   [741 followers]  Follow
    
   Full-text available via subscription Subscription journal
   ISSN (Print) 1078-8956 - ISSN (Online) 1546-170X
   Published by NPG Homepage  [142 journals]
  • Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces
           polycystic ovary syndrome in adulthood
    • Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome in adulthood

      Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome in adulthood, Published online: 14 May 2018; doi:10.1038/s41591-018-0035-5

      Excess anti-Müllerian hormone during pregnancy results in polycystic ovary syndrome-like phenotypes in female offspring, possibly explaining its pathogenesis as well as suggesting a possible therapy.Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome in adulthood, Published online: 2018-05-14; doi:10.1038/s41591-018-0035-52018-05-14
      DOI: 10.1038/s41591-018-0035-5
       
  • CAR T cells for childhood diffuse midline gliomas
    • CAR T cells for childhood diffuse midline gliomas

      CAR T cells for childhood diffuse midline gliomas, Published online: 07 May 2018; doi:10.1038/s41591-018-0031-9

      Anti-GD2 chimeric antigen receptor (CAR)-modified T cells may be a new and innovative approach for the treatment of pediatric H3-K27M-mutant diffuse midline gliomas.CAR T cells for childhood diffuse midline gliomas, Published online: 2018-05-07; doi:10.1038/s41591-018-0031-92018-05-07
      DOI: 10.1038/s41591-018-0031-9
       
  • Sleeper cells
    • Sleeper cells

      Sleeper cells, Published online: 07 May 2018; doi:10.1038/s41591-018-0039-1

      New studies question how HIV persists in the bodySleeper cells, Published online: 2018-05-07; doi:10.1038/s41591-018-0039-12018-05-07
      DOI: 10.1038/s41591-018-0039-1
       
  • Adrenergic nerve degeneration in bone marrow drives aging of the
           hematopoietic stem cell niche
    • Adrenergic nerve degeneration in bone marrow drives aging of the hematopoietic stem cell niche

      Adrenergic nerve degeneration in bone marrow drives aging of the hematopoietic stem cell niche, Published online: 07 May 2018; doi:10.1038/s41591-018-0030-x

      Loss of sympathetic nerve innervention of the bone marrow contributes to the aging of hematopoietic stem cells, which can be rejuvenated using an adrenergic receptor agonist.Adrenergic nerve degeneration in bone marrow drives aging of the hematopoietic stem cell niche, Published online: 2018-05-07; doi:10.1038/s41591-018-0030-x2018-05-07
      DOI: 10.1038/s41591-018-0030-x
       
  • Targeting metabolism to treat psoriasis
    • Targeting metabolism to treat psoriasis

      Targeting metabolism to treat psoriasis, Published online: 07 May 2018; doi:10.1038/s41591-018-0027-5

      A crucial role for glucose metabolism has been identified in wound repair and in the pathogenesis of chronic inflammatory skin diseases, indicating that targeting metabolism is an approach for treating psoriasis.Targeting metabolism to treat psoriasis, Published online: 2018-05-07; doi:10.1038/s41591-018-0027-52018-05-07
      DOI: 10.1038/s41591-018-0027-5
       
  • MC4R agonism promotes durable weight loss in patients with leptin receptor
           deficiency
    • MC4R agonism promotes durable weight loss in patients with leptin receptor deficiency

      MC4R agonism promotes durable weight loss in patients with leptin receptor deficiency, Published online: 07 May 2018; doi:10.1038/s41591-018-0015-9

      Treatment with setmelanotide, a new-generation MC4R agonist, provides durable weight loss in hyperphagic, leptin receptor–deficient patients, suggesting a pharmacological avenue to treat patients with various MC4R pathway defects.MC4R agonism promotes durable weight loss in patients with leptin receptor deficiency, Published online: 2018-05-07; doi:10.1038/s41591-018-0015-92018-05-07
      DOI: 10.1038/s41591-018-0015-9
       
  • Redefining Medicine
    • Redefining Medicine

      Redefining Medicine, Published online: 07 May 2018; doi:10.1038/s41591-018-0037-3

      For more than two decades, Nature Medicine has been the prime venue for publication of outstanding work in the translational space. Now the journal’s scope is evolving to embrace the clinical research that meets the challenges and complexities of contemporary medicine.Redefining Medicine, Published online: 2018-05-07; doi:10.1038/s41591-018-0037-32018-05-07
      DOI: 10.1038/s41591-018-0037-3
       
  • ARID1A deficiency promotes mutability and potentiates therapeutic
           antitumor immunity unleashed by immune checkpoint blockade
    • ARID1A deficiency promotes mutability and potentiates therapeutic antitumor immunity unleashed by immune checkpoint blockade

      ARID1A deficiency promotes mutability and potentiates therapeutic antitumor immunity unleashed by immune checkpoint blockade, Published online: 07 May 2018; doi:10.1038/s41591-018-0012-z

      Loss of mismatch-repair protein ARID1A in cancer correlates with high mutation load & checkpoint blockade response, complementing MSI-based prognosis.ARID1A deficiency promotes mutability and potentiates therapeutic antitumor immunity unleashed by immune checkpoint blockade, Published online: 2018-05-07; doi:10.1038/s41591-018-0012-z2018-05-07
      DOI: 10.1038/s41591-018-0012-z
       
 
 
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