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Journal Cover   Nature Reviews Neuroscience
  [SJR: 14.685]   [H-I: 259]   [191 followers]  Follow
    
   Full-text available via subscription Subscription journal
   ISSN (Print) 1471-003X - ISSN (Online) 1471-0048
   Published by Nature Publishing Group Homepage  [110 journals]
  • Computational neuroscience: Population coupling
    • Authors: Sian Lewis
      Pages: 313 - 313
      Abstract: How the firing of neurons in a population generates cortical representations has not been fully elucidated, but a new study shows that population activity can be understood in terms of the degree of coupling of the spiking of a single neuron to the spiking of the overall population.
      Citation: Nature Reviews Neuroscience 16, 313 (2015)
      PubDate: 2015-04-29
      DOI: 10.1038/nrn3965
      Issue No: Vol. 16, No. 6 (2015)
       
  • Gene expression: Converging pathways
    • Authors: Fiona Carr
      Pages: 314 - 315
      Abstract: Reelin induces transcriptional changes in neurons by increasing the formation of an intracellular fragment of its target receptor that interacts with specific enhancer regions in the nucleus.
      Citation: Nature Reviews Neuroscience 16, 314 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3968
      Issue No: Vol. 16, No. 6 (2015)
       
  • Myelination: An active process
    • Authors: Katherine Whalley
      Pages: 314 - 315
      Abstract: Two studies use zebrafish models to investigate the mechanisms through which neuronal activity influences myelination.
      Citation: Nature Reviews Neuroscience 16, 314 (2015)
      PubDate: 2015-04-29
      DOI: 10.1038/nrn3964
      Issue No: Vol. 16, No. 6 (2015)
       
  • Behavioural neuroscience: Picking up the pups
    • Authors: Natasha Bray
      Pages: 315 - 315
      Abstract: Oxytocin tunes the responses of neurons in the left auditory cortex to pup calls and may thus promote pup retrieval by virgin female mice.
      Citation: Nature Reviews Neuroscience 16, 315 (2015)
      PubDate: 2015-05-08
      DOI: 10.1038/nrn3967
      Issue No: Vol. 16, No. 6 (2015)
       
  • Neural circuits: Pain or pleasure'
    • Authors: Katherine Whalley
      Pages: 316 - 316
      Abstract: A study uses optogenetics to dissect the circuits involved in associating environmental stimuli with positive or negative outcomes.
      Citation: Nature Reviews Neuroscience 16, 316 (2015)
      PubDate: 2015-05-20
      DOI: 10.1038/nrn3975
      Issue No: Vol. 16, No. 6 (2015)
       
  • Glia: The devil in the detail
    • Authors: Sian Lewis
      Pages: 316 - 316
      Abstract: The importance of astrocytic Ca2+ fluctuations in synaptic function has been challenged by the finding that mice devoid of inositol-1,4,5-triphosphate type 2 receptors (IP3R2s) — which are enriched in astrocytes — show normal neuronal and vascular responses. Although changes in intracellular Ca2+
      Citation: Nature Reviews Neuroscience 16, 316 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3973
      Issue No: Vol. 16, No. 6 (2015)
       
  • Cognitive neuroscience: Gridlock
    • Authors: Sian Lewis
      Pages: 316 - 316
      Abstract: Representations of large-scale spaces by grid cells in the medial entorhinal cortex (mEC) requires uniform firing patterns throughout the space being navigated. However, boundaries can disrupt grid-cell firing patterns, suggesting an influence of local environmental cues. Recordings from mEC neurons in a two-compartment chamber showed
      Citation: Nature Reviews Neuroscience 16, 316 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3972
      Issue No: Vol. 16, No. 6 (2015)
       
  • Neurodegeneration: Neurodegeneration takes its TOLL
    • Authors: Sian Lewis
      Pages: 316 - 316
      Abstract: Sterile-α and TIR motif-containing protein 1 (SARM1) is an essential mediator of axon degeneration, and increased levels of NAD+ are neuroprotective, but the link between the two remains unclear. A recent paper showed that, in mice, dimerization of the Toll–interleukin receptor (TIR) domain
      Citation: Nature Reviews Neuroscience 16, 316 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3971
      Issue No: Vol. 16, No. 6 (2015)
       
  • Glia: Diffusible danger
    • Authors: Sian Lewis
      Pages: 316 - 316
      Abstract: The growth of high-grade glioma (HGG) and proliferation of glial precursor cells are enhanced by neuronal activity, but the mechanisms are not known. A new study shows that optogenetic stimulation of cortical neurons in a patient-derived glioblastoma xenograft model in mice increased precursor proliferation. Crucially,
      Citation: Nature Reviews Neuroscience 16, 316 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3970
      Issue No: Vol. 16, No. 6 (2015)
       
  • Neuronal circuits for fear and anxiety
    • Authors: Philip Tovote, Jonathan Paul Fadok, Andreas Lüthi
      Pages: 317 - 331
      Abstract: Decades of research has identified the brain areas that are involved in fear, fear extinction, anxiety and related defensive behaviours. Newly developed genetic and viral tools, optogenetics and advanced in vivo imaging techniques have now made it possible to characterize the activity, connectivity and
      Citation: Nature Reviews Neuroscience 16, 317 (2015)
      PubDate: 2015-05-20
      DOI: 10.1038/nrn3945
      Issue No: Vol. 16, No. 6 (2015)
       
  • Epigenetic and transgenerational reprogramming of brain development
    • Authors: Tracy L. Bale
      Pages: 332 - 344
      Abstract: Neurodevelopmental programming — the implementation of the genetic and epigenetic blueprints that guide and coordinate normal brain development — requires tight regulation of transcriptional processes. During prenatal and postnatal time periods, epigenetic processes fine-tune neurodevelopment towards an end product that determines how an organism interacts
      Citation: Nature Reviews Neuroscience 16, 332 (2015)
      PubDate: 2015-04-29
      DOI: 10.1038/nrn3818
      Issue No: Vol. 16, No. 6 (2015)
       
  • Compromised autophagy and neurodegenerative diseases
    • Authors: Fiona M. Menzies, Angeleen Fleming, David C. Rubinsztein
      Pages: 345 - 357
      Abstract: Most neurodegenerative diseases that afflict humans are associated with the intracytoplasmic deposition of aggregate-prone proteins in neurons and with mitochondrial dysfunction. Autophagy is a powerful process for removing such proteins and for maintaining mitochondrial homeostasis. Over recent years, evidence has accumulated to demonstrate that upregulation
      Citation: Nature Reviews Neuroscience 16, 345 (2015)
      PubDate: 2015-05-20
      DOI: 10.1038/nrn3961
      Issue No: Vol. 16, No. 6 (2015)
       
  • Immune attack: the role of inflammation in Alzheimer disease
    • Authors: Frank L. Heppner, Richard M. Ransohoff, Burkhard Becher
      Pages: 358 - 372
      Abstract: The past two decades of research into the pathogenesis of Alzheimer disease (AD) have been driven largely by the amyloid hypothesis; the neuroinflammation that is associated with AD has been assumed to be merely a response to pathophysiological events. However, new data from preclinical and
      Citation: Nature Reviews Neuroscience 16, 358 (2015)
      PubDate: 2015-05-20
      DOI: 10.1038/nrn3880
      Issue No: Vol. 16, No. 6 (2015)
       
  • Weeding out bad waves: towards selective cannabinoid circuit control in
           epilepsy
    • Authors: Ivan Soltesz, Bradley E. Alger, Masanobu Kano, Sang-Hun Lee, David M. Lovinger, Takako Ohno-Shosaku, Masahiko Watanabe
      Pages: 372 - 372
      Abstract: Nature Reviews Neuroscience16, 264–277 (2015)In Box 2 of this article, the sentence “In FXS, a polyglutamine (CGG) expansion in the nuclear fragile X mental retardation 1 (Fmr1) gene results in its methylation and silencing which
      Citation: Nature Reviews Neuroscience 16, 372 (2015)
      PubDate: 2015-05-13
      DOI: 10.1038/nrn3974
      Issue No: Vol. 16, No. 6 (2015)
       
  • Astrocyte barriers to neurotoxic inflammation
    • Authors: Michael V. Sofroniew
      Pages: 372 - 372
      Abstract: Nature Reviews Neuroscience16, 249–263 (2015)In Figure 3 (left panel) of this article, the interleukins (ILs) being released from the astrocyte functional barrier into the perivascular space (PVS) and subarachnoid space (SAS) should have been listed as IL-1β,
      Citation: Nature Reviews Neuroscience 16, 372 (2015)
      PubDate: 2015-05-08
      DOI: 10.1038/nrn3966
      Issue No: Vol. 16, No. 6 (2015)
       
 
 
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