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Nature Neuroscience
Journal Prestige (SJR): 15.207
Citation Impact (citeScore): 15
Number of Followers: 413  
 
  Full-text available via subscription Subscription journal
ISSN (Print) 1097-6256 - ISSN (Online) 1546-1726
Published by NPG Homepage  [144 journals]
  • Author Correction: Transneuronal propagation of mutant huntingtin
           contributes to non–cell autonomous pathology in neurons
    • Author Correction: Transneuronal propagation of mutant huntingtin contributes to non–cell autonomous pathology in neurons

      Author Correction: Transneuronal propagation of mutant huntingtin contributes to non–cell autonomous pathology in neurons, Published online: 17 July 2018; doi:10.1038/s41593-018-0201-6

      Author Correction: Transneuronal propagation of mutant huntingtin contributes to non–cell autonomous pathology in neuronsAuthor Correction: Transneuronal propagation of mutant huntingtin contributes to non–cell autonomous pathology in neurons, Published online: 2018-07-17; doi:10.1038/s41593-018-0201-62018-07-17
      DOI: 10.1038/s41593-018-0201-6
       
  • Author Correction: Dopamine transients are sufficient and necessary for
           acquisition of model-based associations
    • Author Correction: Dopamine transients are sufficient and necessary for acquisition of model-based associations

      Author Correction: Dopamine transients are sufficient and necessary for acquisition of model-based associations, Published online: 17 July 2018; doi:10.1038/s41593-018-0202-5

      Author Correction: Dopamine transients are sufficient and necessary for acquisition of model-based associationsAuthor Correction: Dopamine transients are sufficient and necessary for acquisition of model-based associations, Published online: 2018-07-17; doi:10.1038/s41593-018-0202-52018-07-17
      DOI: 10.1038/s41593-018-0202-5
       
  • Publisher Correction: Tuned thalamic excitation is amplified by visual
           cortical circuits
    • Publisher Correction: Tuned thalamic excitation is amplified by visual cortical circuits

      Publisher Correction: Tuned thalamic excitation is amplified by visual cortical circuits, Published online: 16 July 2018; doi:10.1038/s41593-018-0181-6

      Publisher Correction: Tuned thalamic excitation is amplified by visual cortical circuitsPublisher Correction: Tuned thalamic excitation is amplified by visual cortical circuits, Published online: 2018-07-16; doi:10.1038/s41593-018-0181-62018-07-16
      DOI: 10.1038/s41593-018-0181-6
       
  • PKCα integrates spatiotemporally distinct Ca2+ and autocrine BDNF
           signaling to facilitate synaptic plasticity
    • PKCα integrates spatiotemporally distinct Ca2+ and autocrine BDNF signaling to facilitate synaptic plasticity

      PKCα integrates spatiotemporally distinct Ca2+ and autocrine BDNF signaling to facilitate synaptic plasticity, Published online: 16 July 2018; doi:10.1038/s41593-018-0184-3

      Through the development of novel PKC biosensors, the authors describe how PKCα, but not other classical isozymes, facilitates plasticity in dendritic regions through the integration of recent synaptic plasticity with current, local synaptic input.PKCα integrates spatiotemporally distinct Ca2+ and autocrine BDNF signaling to facilitate synaptic plasticity, Published online: 2018-07-16; doi:10.1038/s41593-018-0184-32018-07-16
      DOI: 10.1038/s41593-018-0184-3
       
  • Author Correction: PV plasticity sustained through D1/5 dopamine signaling
           required for long-term memory consolidation
    • Author Correction: PV plasticity sustained through D1/5 dopamine signaling required for long-term memory consolidation

      Author Correction: PV plasticity sustained through D1/5 dopamine signaling required for long-term memory consolidation, Published online: 16 July 2018; doi:10.1038/s41593-018-0179-0

      Author Correction: PV plasticity sustained through D1/5 dopamine signaling required for long-term memory consolidationAuthor Correction: PV plasticity sustained through D1/5 dopamine signaling required for long-term memory consolidation, Published online: 2018-07-16; doi:10.1038/s41593-018-0179-02018-07-16
      DOI: 10.1038/s41593-018-0179-0
       
  • FRETting over postsynaptic PKC signaling
    • FRETting over postsynaptic PKC signaling

      FRETting over postsynaptic PKC signaling, Published online: 16 July 2018; doi:10.1038/s41593-018-0190-5

      Protein kinases are key regulators of excitatory synapse plasticity. In this issue, using novel optical reporters of protein kinase C (PKC) activity, Colgan et al. identify PKCα as critical for integrating NMDA receptor and neurotrophin signaling to control dendritic spine structural plasticity, synaptic potentiation, and learning and memory.FRETting over postsynaptic PKC signaling, Published online: 2018-07-16; doi:10.1038/s41593-018-0190-52018-07-16
      DOI: 10.1038/s41593-018-0190-5
       
  • The maternal vaginal microbiome partially mediates the effects of prenatal
           stress on offspring gut and hypothalamus
    • The maternal vaginal microbiome partially mediates the effects of prenatal stress on offspring gut and hypothalamus

      The maternal vaginal microbiome partially mediates the effects of prenatal stress on offspring gut and hypothalamus, Published online: 09 July 2018; doi:10.1038/s41593-018-0182-5

      Maternal stress during pregnancy is a risk factor for neurodevelopmental disorders. Jasarevic and colleagues show that the maternal vaginal microbiota partially mediates the lasting effects of prenatal stress on the gut and hypothalamus in mice.The maternal vaginal microbiome partially mediates the effects of prenatal stress on offspring gut and hypothalamus, Published online: 2018-07-09; doi:10.1038/s41593-018-0182-52018-07-09
      DOI: 10.1038/s41593-018-0182-5
       
  • Author Correction: Hippocampus-driven feed-forward inhibition of the
           prefrontal cortex mediates relapse of extinguished fear
    • Author Correction: Hippocampus-driven feed-forward inhibition of the prefrontal cortex mediates relapse of extinguished fear

      Author Correction: Hippocampus-driven feed-forward inhibition of the prefrontal cortex mediates relapse of extinguished fear, Published online: 09 July 2018; doi:10.1038/s41593-018-0183-4

      Author Correction: Hippocampus-driven feed-forward inhibition of the prefrontal cortex mediates relapse of extinguished fearAuthor Correction: Hippocampus-driven feed-forward inhibition of the prefrontal cortex mediates relapse of extinguished fear, Published online: 2018-07-09; doi:10.1038/s41593-018-0183-42018-07-09
      DOI: 10.1038/s41593-018-0183-4
       
 
 
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