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Journal Cover Nature Genetics
  [SJR: 23.762]   [H-I: 469]   [516 followers]  Follow
    
   Full-text available via subscription Subscription journal
   ISSN (Print) 1061-4036 - ISSN (Online) 1546-1718
   Published by NPG Homepage  [135 journals]
  • Evolutionary analysis indicates that DNA alkylation damage is a byproduct
           of cytosine DNA methyltransferase activity
    • Evolutionary analysis indicates that DNA alkylation damage is a byproduct of cytosine DNA methyltransferase activity

      Evolutionary analysis indicates that DNA alkylation damage is a byproduct of cytosine DNA methyltransferase activity, Published online: 19 February 2018; doi:10.1038/s41588-018-0061-8

      The authors report that DNA methylation coevolves with the DNA alkylation repair enzyme ALKB2 across eukaryotes. They also show that DNA methyltransferases cause alkylation damage in vitro and in vivo by introducing 3-methylcytosine into DNA.Evolutionary analysis indicates that DNA alkylation damage is a byproduct of cytosine DNA methyltransferase activity, Published online: 2018-02-19; doi:10.1038/s41588-018-0061-82018-02-19
      DOI: 10.1038/s41588-018-0061-8
       
  • Genome-wide mapping of global-to-local genetic effects on human facial
           shape
    • Genome-wide mapping of global-to-local genetic effects on human facial shape

      Genome-wide mapping of global-to-local genetic effects on human facial shape, Published online: 19 February 2018; doi:10.1038/s41588-018-0057-4

      The authors report a data-driven approach to phenotyping 3D facial shape. They apply their methodology to 2,329 individuals of European ancestry and identify 38 loci that associate with specific facial morphologies, some of which overlap with neural-crest-specific regulatory regions.Genome-wide mapping of global-to-local genetic effects on human facial shape, Published online: 2018-02-19; doi:10.1038/s41588-018-0057-42018-02-19
      DOI: 10.1038/s41588-018-0057-4
       
  • Embryonic defects induced by maternal obesity in mice derive from Stella
           insufficiency in oocytes
    • Embryonic defects induced by maternal obesity in mice derive from Stella insufficiency in oocytes

      Embryonic defects induced by maternal obesity in mice derive from Stella insufficiency in oocytes, Published online: 19 February 2018; doi:10.1038/s41588-018-0055-6

      The authors find that, in a high-fat diet (HFD) mouse model, levels of Stella protein are reduced in oocytes, leading to abnormal epigenetic patterning during development and to embryonic growth defects. Overexpression of Stella in oocytes from HFD-fed mice partially ameliorates developmental defects.Embryonic defects induced by maternal obesity in mice derive from Stella insufficiency in oocytes, Published online: 2018-02-19; doi:10.1038/s41588-018-0055-62018-02-19
      DOI: 10.1038/s41588-018-0055-6
       
  • Meta-analysis of genome-wide association studies for cattle stature
           identifies common genes that regulate body size in mammals
    • Meta-analysis of genome-wide association studies for cattle stature identifies common genes that regulate body size in mammals

      Meta-analysis of genome-wide association studies for cattle stature identifies common genes that regulate body size in mammals, Published online: 19 February 2018; doi:10.1038/s41588-018-0056-5

      Meta-analysis of data from 58,265 cattle shows that the genetic architecture underlying stature is similar to that in humans, where many genomic regions individually explain only a small amount of phenotypic variance.Meta-analysis of genome-wide association studies for cattle stature identifies common genes that regulate body size in mammals, Published online: 2018-02-19; doi:10.1038/s41588-018-0056-52018-02-19
      DOI: 10.1038/s41588-018-0056-5
       
  • A CRISPR-based screen for Hedgehog signaling provides insights into
           ciliary function and ciliopathies
    • A CRISPR-based screen for Hedgehog signaling provides insights into ciliary function and ciliopathies

      A CRISPR-based screen for Hedgehog signaling provides insights into ciliary function and ciliopathies, Published online: 19 February 2018; doi:10.1038/s41588-018-0054-7

      A CRISPR-based functional screen for Hedgehog-pathway factors identifies genes required for ciliary signaling and can be used to classify genetic disorders as ciliopathies.A CRISPR-based screen for Hedgehog signaling provides insights into ciliary function and ciliopathies, Published online: 2018-02-19; doi:10.1038/s41588-018-0054-72018-02-19
      DOI: 10.1038/s41588-018-0054-7
       
  • Publisher Correction: BRD4 interacts with NIPBL and BRD4 is mutated in a
           Cornelia de Lange–like syndrome
    • Publisher Correction: BRD4 interacts with NIPBL and BRD4 is mutated in a Cornelia de Lange–like syndrome

      Publisher Correction: BRD4 interacts with NIPBL and BRD4 is mutated in a Cornelia de Lange–like syndrome, Published online: 12 February 2018; doi:10.1038/s41588-018-0069-0

      Publisher Correction: BRD4 interacts with NIPBL and BRD4 is mutated in a Cornelia de Lange–like syndromePublisher Correction: BRD4 interacts with NIPBL and BRD4 is mutated in a Cornelia de Lange–like syndrome, Published online: 2018-02-12; doi:10.1038/s41588-018-0069-02018-02-12
      DOI: 10.1038/s41588-018-0069-0
       
  • Stress and sexual reproduction affect the dynamics of the wheat pathogen
           effector AvrStb6 and strobilurin resistance
    • Stress and sexual reproduction affect the dynamics of the wheat pathogen effector AvrStb6 and strobilurin resistance

      Stress and sexual reproduction affect the dynamics of the wheat pathogen effector AvrStb6 and strobilurin resistance, Published online: 12 February 2018; doi:10.1038/s41588-018-0052-9

      Identification of AvrStb6, the fungal avirulence effector that triggers Stb6-mediated resistance in wheat, here demonstrates that neither host resistance nor fungicide treatment suppresses fungal sexual reproduction, thus unveiling implications of fungal sex in plant disease control.Stress and sexual reproduction affect the dynamics of the wheat pathogen effector AvrStb6 and strobilurin resistance, Published online: 2018-02-12; doi:10.1038/s41588-018-0052-92018-02-12
      DOI: 10.1038/s41588-018-0052-9
       
  • Wheat receptor-kinase-like protein Stb6 controls gene-for-gene resistance
           to fungal pathogen Zymoseptoria tritici
    • Wheat receptor-kinase-like protein Stb6 controls gene-for-gene resistance to fungal pathogen Zymoseptoria tritici

      Wheat receptor-kinase-like protein Stb6 controls gene-for-gene resistance to fungal pathogen Zymoseptoria tritici, Published online: 12 February 2018; doi:10.1038/s41588-018-0051-x

      The authors report map-based cloning of the wheat Stb6 gene, which encodes a conserved wall-associated receptor kinase (WAK)-like protein. Stb6 confers gene-for-gene disease resistance to fungal pathogen Zymoseptoria tritici by recognition of a matching pathogen effector.Wheat receptor-kinase-like protein Stb6 controls gene-for-gene resistance to fungal pathogen Zymoseptoria tritici, Published online: 2018-02-12; doi:10.1038/s41588-018-0051-x2018-02-12
      DOI: 10.1038/s41588-018-0051-x
       
 
 
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